LTP | vesicle-inducing protein in plastids (VIPP1)

Name: LTP | vesicle-inducing protein in plastids (VIPP1)
SKU: AS06 145
Price: 302 EUR
Availability: InStock
Rating: 5 (1 reviews)

AS06 145 | Clonality: Polyclonal | Host: Rabbit | Reactivity: Arabidopsis thaliana, Chlamydomonas reinhardtii

LTP | vesicle-inducing protein in plastids (VIPP1) in the group Antibodies Plant/Algal / Photosynthesis / Chaperones/Transporters at Agrisera AB (Antibodies for research) (AS06 145)

LTP | vesicle-inducing protein in plastids (VIPP1)

Product Information

Immunogen

Recombinant VIPP1 of Chlamydomonas reinhardtii, UniProt: Q66YD0

Host Rabbit

Clonality Polyclonal

Purity Serum

Format Lyophilized

Quantity 200 µl

Reconstitution For reconstitution add 200 µl of sterile water

Storage Store lyophilized/reconstituted at -20°C; once reconstituted make aliquots to avoid repeated freeze-thaw cycles. Please remember to spin the tubes briefly prior to opening them to avoid any losses that might occur from material adhering to the cap or sides of the tube.

Tested applications Western blot (WB)

Recommended dilution 1 : 2000 (WB)

Expected | apparent MW

34 kDa

Reactivity

Confirmed reactivity Arabidopsis thaliana, Chlamydomonas reinhardtii

Predicted reactivity

Synechococcus sp.

Species of your interest not listed? Contact us

Not reactive in Pheodactylum tricornutum

Application examples

Additional information

Background

VIPP1 - vesicle-inducing protein in plastids 1, is a hydrophilic found in both the inner envelope and the thylakoid membranes. In VIPP1 deletion mutants vesicle formation is abolished. VIPP1 is proposed to be essential for the maintenance of thylakoids.

Product citations

Selected references Jeran et al. (2021) The PUB4 E3 Ubiquitin Ligase Is Responsible for the Variegated Phenotype Observed upon Alteration of Chloroplast Protein Homeostasis in Arabidopsis Cotyledons. Genes (Basel). 2021 Sep 6;12(9):1387. doi: 10.3390/genes12091387. PMID: 34573369; PMCID: PMC8464772.
Fristedt et al. (2015). The thylakoid membrane protein CGL160 supports CF1CF0 ATP synthase accumulation in Arabidopsis thaliana. PLoS One. 2015 Apr 2;10(4):e0121658. doi: 10.1371/journal.pone.0121658.
Kroll et al. (2001) VIPP1, a nuclear gene of Arabidopsis thaliana essential for thylakoid membrane formation. Proc Natl Acad Sci U S A 98: 4238-4242.

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immunogen:	Recombinant VIPP1 of Chlamydomonas reinhardtii, UniProt: Q66YD0
Reconstitution:	For reconstitution add 200 µl of sterile water
Host:	Rabbit
Clonality:	Polyclonal
Purity:	Serum
Format:	Lyophilized
Quantity:	200 µl
storage:	Store lyophilized/reconstituted at -20°C; once reconstituted make aliquots to avoid repeated freeze-thaw cycles. Please remember to spin the tubes briefly prior to opening them to avoid any losses that might occur from material adhering to the cap or sides of the tube.
tested applications:	Western blot (WB)
recommended dilution:	1 : 2000 (WB)
calculated \| apparent molecular mass [kDa]:	34 kDa
Confirmed reactivity:	Arabidopsis thaliana, Chlamydomonas reinhardtii
predicted reactivity:	Synechococcus sp. Species of your interest not listed? Contact us
not reactive in:	Pheodactylum tricornutum
background:	VIPP1 - vesicle-inducing protein in plastids 1, is a hydrophilic found in both the inner envelope and the thylakoid membranes. In VIPP1 deletion mutants vesicle formation is abolished. VIPP1 is proposed to be essential for the maintenance of thylakoids.
All references:	Jeran et al. (2021) The PUB4 E3 Ubiquitin Ligase Is Responsible for the Variegated Phenotype Observed upon Alteration of Chloroplast Protein Homeostasis in Arabidopsis Cotyledons. Genes (Basel). 2021 Sep 6;12(9):1387. doi: 10.3390/genes12091387. PMID: 34573369; PMCID: PMC8464772. Fristedt et al. (2015). The thylakoid membrane protein CGL160 supports CF1CF0 ATP synthase accumulation in Arabidopsis thaliana. PLoS One. 2015 Apr 2;10(4):e0121658. doi: 10.1371/journal.pone.0121658. Kroll et al. (2001) VIPP1, a nuclear gene of Arabidopsis thaliana essential for thylakoid membrane formation. Proc Natl Acad Sci U S A 98: 4238-4242.